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. Author manuscript; available in PMC: 2009 Jul 11.
Published in final edited form as: Mol Cell. 2008 Jul 11;31(1):21–32. doi: 10.1016/j.molcel.2008.04.028

Table 1. Analysis of p53 and p19ARF sequence in tumor samples.

Exons 5–10 of p53 genomic DNA and exons 2–11 of p53 cDNA were PCR amplified and sequenced. The coding sequence of p19ARF was amplified and sequenced (exons 1–3) from cDNA samples. All sequencing was performed multiple times and PCR products were sequenced in both directions.

p53 exon amplification from genomic DNA
ID genotype tumor type Ex. 5 Ex. 6 Ex.7 Ex. 8 Ex. 9 Ex. 10
22790 M-C2 basal carcinoma WT WT WT WT WT WT
23186 N-C2 adeno/squamous carcinoma WT WT WT WT WT WT
23182 M-C2 osteosarcoma WT WT WT WT WT WT
23828 N-C2 granulosa cell tumor WT WT WT WT WT WT
21872 N-C2 histiocytic sarcoma WT WT WT WT WT WT
23646 N-C2 lymphoma WT WT WT WT WT WT
23649 N-C2 mammary carcinoma WT WT WT WT WT ND
24098 N-C2 basal carcinoma WT WT WT WT WT WT
p53 and p19ARF amplification from cDNA
ID tumor type p53 Cdna
exons 2–11
p19ARF cDNA
exons 1–3
23902 M-C2 basal carcinoma WT NA
24071 N-C2 lymphoma WT NA
23828 N-C2 granulosa cell tumor WT WT
23129 M-C2 histiocytic sarcoma WT WT
23186 N-C2 adeno/squamous carcinoma WT WT
25094 N-C2 uterine leiomyosarcoma WT WT
23646 N-C2 lymphoma WT WT

(M-C2=Mre11ATLD1/ATLD1 Chk2−/−, N-C2=Nbs1ΔB/ΔB Chk2−/−, WT=wild type, ND=not determined, NA=not amplifiable)