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Increased formation of tissue NO metabolites from
during acute global hypoxia in
vivo. Blood and tissues utilize
during global hypoxia in
vivo (A) to generate NO-related products including
S-nitrosothiols (RSNO) (B), N-nitrosamines (RNNO)
(C), and iron nitrosyls (NO-heme) (D). (n = 3); *,
p < 0.05 versus normoxia.
