. Author manuscript; available in PMC: 2009 Oct 15.

Published in final edited form as: Dev Biol. 2008 Aug 7;322(2):322–331. doi: 10.1016/j.ydbio.2008.07.035

Table 4.

skr-1(sm151) suppresses lin-12(rf) in a sel-10(n1074) background

genotype	% 2 AC phenotype (25°C)	n
skr-1(sm151)	0	100
skr-1(sm151); sel-10(n1074)	0	100
skr-1(sm151); sel-10(ok1632)	0	100
lin-12(ar170)	80	100
skr-1(sm151); lin-12(ar170)	77	100
lin-12(ar170); sel-10(n1074)	25^*	200
lin-12(ar170); sel-10(ok1632)	19	100
skr-1(sm151); lin-12(ar170); sel-10(n1074)	18^*	200
skr-1(sm151); lin-12(ar170); sel-10(ok1632)	18	100
skr-1(sm151); unc-76	0	100
skr-1(sm151); sel-10(ar41) unc-76	0	100
lin-12(ar170); unc-76	79	100
skr-1(sm151); lin-12(ar170); unc-76	81	100
lin-12(ar170); sel-10(ar41) unc-76	21	100
skr-1(sm151); lin-12(ar170); sel-10(ar41) unc-76	19	100

ACs were scored with DIC (100x) at the late L3/early L4 stage.

These values are statistically different based on Chi square analysis, P < 0.03. unc-76(e911) is linked to sel-10(ar41) and was included in relevant control strains.