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. 2007 Oct 31;48(5):847–855. doi: 10.3349/ymj.2007.48.5.847

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Copyright © 2007 The Yonsei University College of Medicine

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0) which permits unrestricted noncommercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 5 — Effects of vitamin C on apoptosis in diabetic rats. Intranucleosomal DNA fragmentation was labeled using the Apoptosis detection system after 16 weeks of vitamin C treatment, as described in Materials and Methods. D, diabetic rats (A); D + VC, diabetic rats treated with vitamin C (B). Magnification, × 100. Apoptosis positive cells were counted in sixconsecutive interstitial fields under the microscope and expressed as the mean numbers of positive cells of the interstitial field (C). Values are means ± SD. D, diabetic rats; D + VC, diabetic rats treated with vitamin C. ^*p < 0.05 compared with D.