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. Author manuscript; available in PMC: 2009 Jan 18.
Published in final edited form as: Science. 2006 Jun 1;313(5783):63–68. doi: 10.1126/science.1128650

Fig. 5.

Fig. 5

Contributions of dpp transcription and mobility to growth. (A and B) dpp-lacZ is up-regulated in Ubx mutant haltere tissue (marked by loss of GFP) within the AP organizer. (C and D) UAS-tkv+ expression in dorsal cells using ap-Gal4 results in dpp-lacZ down-regulation (thin arrow) compared with ventral cells (thick arrow). (E to G) Examples of +/Ubx- (E), pbx/Ubx- (F), and pbx/Ubx-; actGal4>tkv+ (G) haltere discs stained for Nubbin (Nub), a marker of the appendage, and a marker of the AP compartment boundary (yellow line). (H)P:A ratios of the Nub domains of +/Ubx-, pbx/Ubx-, and pbx/Ubx-; actGal4>tkv+ haltere + discs. Error bars indicate SEM. (I) Summary of the consequences of different spatial relationships of Dpp and Hh signaling for dpp transcription in the wing and haltere. (J) Summary of how Dpp represses mtv in the presence of Ubx to control tkv expression, Dpp mobility, and growth in the haltere compared with the wing.