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. Author manuscript; available in PMC: 2009 Dec 15.
Published in final edited form as: J Immunol. 2008 Dec 15;181(12):8670–8676. doi: 10.4049/jimmunol.181.12.8670

Figure 4. CCR1-deficiency does not alter extent of renal dysfunction after ischemia-reperfusion injury.

Figure 4

A, Histopathologic analysis. PAS-stained outer medulla of the left (injured) kidney is shown for wild type (Ai, iii, v, vii, ix) and CCR1-deficient (Aii, iv, vi, viii) mice at the times after ischemia-reperfusion injury indicated to the left of each panel (Ai-viii) or 24 hours after sham operation (ix). Scale bar indicates 50µm. B–E, Quantitation of the indicated measures of renal damage post-IR. sham, sham-operated wild type mice tested 24 hours after surgery. Values are mean ± SEM and are from 3 independent experiments with 3–5 animals for each condition in each experiment.