Table 1.

Shared and differentiated functions among the dominantly mutated Aux/IAA proteins

Phenotype	Degree of Defects					Target Auxin Response Factors (ARF) (Putative)
	Dominant Mutants	Driven by MSG2 Promoter		Driven by AXR2 Promoter
Size of nature plants	wt1) = msg2 < slr < axr2	wt =	msg2 = slr = axr2*	wt =	msg2 < slr = axr2	ARF6 + ARF818
Embryogenesis	wt = msg2 = slr = axr2 +NPH4/ARF716	wt =	msg2 < slr = axr2	wt =	msg2 = slr = axr2*	MP/ARF5
Shape of etiolated hypocotyls	wt ≈ msg2 < slr, axr22)	wt ≈	msg2 = slr = axr2	wt ≈	msg2 < slr, axr22)	ARF6 + ARF818
Hypocotyl gravitropism	wt < msg2 < slr < axr2	wt <	msg2< slr = axr2	wt <	msg2< slr = axr2	NPH4 + ARF1919,20
Lateral root formation	wt < msg2, axr22) < slr	wt <	msg2 = slr = axr2	ND1)		NPH4 + ARF1920,21
Root gravitropism	wt = msg2 < slr = axr2	wt =	msg2 = slr = axr2*	ND		NPH4 + ARF1920
Root hair formation	wt = msg2 < slr = axr2	wt =	msg2 = slr = axr2*	ND		?

^*This shows that MSG2 or AXR2 does not express in a tissue critical for the phenotype. Proteins appearing in a black background show the same function among the mutated Aux/IAA proteins; those in a grey background show quantitative differences in function among them; underlined names show qualitative differences;

¹⁾wt, wild type; ND, not determined;

²⁾Two mutants exhibit qualitatively different phenotypes.