Neonatal exposure to alcohol results in reduced spontaneous motor activity in 27–28 day-old rats (A), with the same trend also present on PD47 (B) (*, p<0.05 between groups). Following gaboxadol treatment (5 mg/kg, s.c.) on PD51–52, motor activity is reduced more profoundly (C), and the latency to gaboxadol-induced transient period of immobility lasting at least 3 min is shorter in the alcohol- than sham-treated rats (D).