Figure 3.

The scheme of potential interactions determining cellular response to oxidative stress. ROS/RNS are formed in the cells as the part of normal metabolism as well as in response to environmental stresses. ROS/RNS themselves as well as growth regulators regulate scavenging pathways to keep ROS/RNS levels low (for simplicity, only auxin is shown as beeing the main growth regulator implicated in cell cycle activation). In case net ROS/RNS production exceeds a limit due to increased production or decreased removal, differentiated cells will die. In contrast, a transient and moderate increase in ROS/RNS level may enhance cell activation in concert with auxin action.