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. 2008 Aug;3(8):537–542. doi: 10.4161/psb.3.8.5748

Figure 4.

Figure 4

GH3.5 modulates SA-dependent defense response. (A) Northern blot analysis of PR-1 induction by exogenous application of SA (0.5 mM) over a time course of 0 to 48 h after treatment in gh3.5-1D heterozygous (gh3.5-1D+/−) and homozygous (gh3.5-1D−/−) plants, in comparison with Col-0 plants. The experiment was biologically repeated once. (B) Expression of PR-1 in NahG, gh3.5-1D/NahG double mutant and gh3.5-1D plants after infection with Psm(avrRpm1) at 107 cfu ml−1. Leaves were collected at 0 and 48 hpi. The experiments were repeated once with similar results. (C) Growth of Pst DC3000 in Col-0, NahG and gh3.5-1D/NahG plants after pre-treatment with SA (1 mM) or buffer (mock). Bacterial titers were repeated twice with similar results. All values are the mean ± SE (n = 6).