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. 2009 Jan 21;106(5):1496–1501. doi: 10.1073/pnas.0802674106

Table 2.

Suppressor screens

Gene Brief description N2,mean ± SEM (N) ctbp-1(ok498),mean ± SEM (N) P value No. of trials
Control 15.1 ± 0.1 (870) 18.2 ± 0.1 (832) <0.0001* 11
AC3.3 Induced by blockage of the UPR in the ER 15.2 ± 0.3 (266) 18.0 ± 0.3 (261) <0.0001*, 0.87 3
C06E8.5 Bactericidal/permeability-increasing protein 15.3 ± 0.2 (295) 18.4 ± 0.2 (223) <0.0001*, 0.63 3
lips-7 Triacylglycerol lipase 15.5 ± 0.3 (154) 15.0 ± 0.3 (151) 0.28*, <0.0001 3
F28F8.2 Long chain fatty acid acyl-CoA ligase 15.2 ± 0.3 (258) 18.6 ± 0.2 (258) <0.0001*, 0.05 3
F47G4.3 Glycerol-3-phosphate dehydrogenase 14.0 ± 0.2 (343) 16.1 ± 0.3 (331) <0.0001*, 0.009 4
K08B12.1 Triacylglycerol lipase 15.3 ± 0.2 (283) 18.5 ± 0.3 (263) <0.0001*, 0.03 3
F10D11.6 Bactericidal/permeability-increasing protein

Knock down of CTBP-1 target genes to examine their roles in life span regulation. Life span results represent pooled data from individual experiments. UPR, unfolded protein response.

*Comparing ctbp-1(ok498) with N2 treated with the same specific RNAi.

Comparing ctbp-1(ok498) treated with the specific RNAi with that treated with control RNAi.

These worms were sick and died very early.