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. Author manuscript; available in PMC: 2009 Aug 12.
Published in final edited form as: Oncogene. 2008 Nov 24;28(6):910–920. doi: 10.1038/onc.2008.428

Figure 2. ER stress induces Akt activity in a PERK-dependent manner.

Figure 2

(A) NIH-3T3 cells or (B) wild type and PERK−/− fibroblasts were treated with 2µg/mL tunicamycin or DMSO as a vehicle control for the indicated intervals. Cell lysates were resolved by SDS-PAGE and membranes were probed with antibodies for phosphorylated and total Akt and eIF2α.