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. 2008 Sep 5;40(3):332–339. doi: 10.1165/rcmb.2008-0003OC

Figure 4.

Figure 4.

WHPC minimizes hypoxia-induced release of type I epithelial markers. (A) Exposure to hypoxia caused a substantial increase in caveolin-2 protein levels in BAL, which was greatly attenuated by pretreatment with WHPC. Shown is a representative Western blot. (B) Densitometric analysis of caveolin-2 immunosignals from five blots. Data are means ± SE; n = 10 for each group. *P < 0.001 versus control; #P < 0.005 versus control; P < 0.01 versus hypoxia. (C) Hypoxia exposure also increased receptor for advanced glycation end products (RAGE) levels in BAL, which was completely abolished by pretreatment with WHPC. Shown is a representative Western blot. (D) Densitometric analysis of RAGE immunosignals from 4 blots. Data are mean ± SE; n = 8 for each group. *P < 0.001 versus control; #P < 0.001 versus hypoxia.