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. Author manuscript; available in PMC: 2009 Feb 24.
Published in final edited form as: J Parasitol. 2008 Dec;94(6):1253–1258. doi: 10.1645/GE-1481.1

Figure 1.

Figure 1

Quantitative levels of Th1 cytokine (IL-2) in (A) granulomas derived from Taenia crassiceps infected, wild type versus SP-Precursor knockout and NK1 receptor knockout mice. *P< 05, IL-2 levels in infected, SP-Precursor knockout-derived granulomas versus wild type-derived granulomas, †P< .05, IL-2 levels in infected, NK1 knockout-derived granulomas versus wild type-derived granulomas. (B) Early versus late granulomas derived from Taenia crassiceps infected, wild type versus SP-Precursor knockout and NK1 receptor knockout mice. *P< 05, IL-2 levels in infected, early stage versus late stage NK1 knockout derived granulomas.