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. 2006 May 25;174(4):461–470. doi: 10.1164/rccm.200512-1886OC

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Figure 5. — Overexpression of p80 accelerates allograft dysfunction. In (A–D), wild-type C57BL/6J (C), Clara cell secretory protein (CCSP)–p40 transgenic C57BL/6J (Tg-p40 C), or IL-12 p35^−/− C57 (p35^−/− C) donor tracheas underwent transplantation into wild-type BALB/cJ (B) or BALB/cJ (Rβ1^−/− B) mice. At 14 or 28 d post-transplantation, grafts underwent H&E staining (A), quantification of the percentage of denuded basement membrane (B), immune cell accumulation (C), and hydroxyproline (D) as described in Figure 1. Values represent means ± SEM (n = 4 or 5). *p < 0.05 compared with C → B.

Inline graphic — Overexpression of p80 accelerates allograft dysfunction. In (A–D), wild-type C57BL/6J (C), Clara cell secretory protein (CCSP)–p40 transgenic C57BL/6J (Tg-p40 C), or IL-12 p35^−/− C57 (p35^−/− C) donor tracheas underwent transplantation into wild-type BALB/cJ (B) or BALB/cJ (Rβ1^−/− B) mice. At 14 or 28 d post-transplantation, grafts underwent H&E staining (A), quantification of the percentage of denuded basement membrane (B), immune cell accumulation (C), and hydroxyproline (D) as described in Figure 1. Values represent means ± SEM (n = 4 or 5). *p < 0.05 compared with C → B.