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. Author manuscript; available in PMC: 2009 Mar 19.
Published in final edited form as: Diabetes Care. 2007 Jul 20;30(10):2613–2618. doi: 10.2337/dc07-0850

Table 1.

Dichotomous clinical and nerve conduction measures used to identify distal symmetrical neuropathy at DCCT baseline and at DCCT completion

DCCT baseline DCCT completion


All subjects Subgroup 1 Subgroup 2 Subgroup 3 Subgroup 4*
Attribute/nerve (n)
  Intensive treatment group 711 647 607 470 361
  Conventional treatment group 730 606 567 330 228
Clinical evidence of distal symmetrical neuropathy
  Symptoms
    Intensive treatment group 5.8 4.2 2.4 2.7 0.3
    Conventional treatment group 6.6 7.5 5.5 7.1 1.4
  Sensory signs
    Intensive treatment group 22.1 18.3 13.6 12.6 0.3
    Conventional treatment group 20.8 21.6 16.2 14.9 1.0
  Decreased reflexes
    Intensive treatment group 18.0 17.8 13.7 11.1 1.2
    Conventional treatment group 15.7 25.9 21.7 15.2 3.4
Nerve conduction abnormality of selected nerve
  Abnormal median motor
    Intensive treatment group 20.9 11.4§ 11.3§ 4.2 3.8
    Conventional treatment group 22.8 24.5 25.3 7.4 7.6
  Abnormal median sensory
    Intensive treatment group 34.1 39.0 39.8 27.0 25.1
    Conventional treatment group 32.0 42.9 44.3 24.0 23.3
  Abnormal peroneal motor
    Intensive treatment group 35.4 23.8§ 24.4§ 11.2 11.2
    Conventional treatment group 42.3 46.1 47.9 19.9 17.1
  Abnormal sural sensory
    Intensive treatment group 28.5 21.4§ 22.2 7.4 6.5
    Conventional treatment group 26.1 31.3 32.4 6.4 5.7

Data are percent, unless otherwise indicated. The dichotomous clinical and nerve conduction results for DCCT completion reflect analyses after sequentially excluding subjects with confirmed clinical neuropathy, the primary DCCT neuropathy end point (subgroup 1); definite clinical neuropathy (subgroup 2); definite clinical neuropathy or possible clinical neuropathy (subgroup 3); and definite clinical neuropathy, possible clinical neuropathy, or subclinicalneuropathy (subgroup 4). Subgroup 4 represents subjects who did not meet any of the DCCT definitions of clinical or subclinical neuropathy.

*

The prevalence of symptoms and signs in this subgroup is not zero because the neurologist concluded that the potential abnormality consistent with distal symmetrical neuropathy had another explanation (e.g., 11 subjects had diffusely hypoactive or absent reflexes without symptoms or other signs of neuropathy).

P < 0.01;

P < 0.001;

§

P < 0.0001.