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. Author manuscript; available in PMC: 2010 Jan 3.
Published in final edited form as: J Neuroimmunol. 2008 Nov 17;206(1-2):22–27. doi: 10.1016/j.jneuroim.2008.10.006

Figure 2.

Figure 2

The clinical course of adoptive transferred EAE is significantly attenuated on transfer of CD11a-/- MOG sensitized T cells to wild type recipients, but exacerbated on transfer of wild type or CD11a-/- MOG-sensitized T cells to CD11a-/- recipients. A, Transferred EAE was induced as described in Materials and Methods in wild-type mice (n=8) by injecting encephalitogenic T cells (2.5 × 107) derived from CD11a-/- mice with active EAE. As a control transferred EAE was induced in wild type mice (n=7) by injecting encephalitogenic T cells (2.5 × 107) derived from wild-type mice with active EAE. B, Transferred EAE was induced as in A, in wild type (n=7) and CD11a-/- mice (n=7) by injecting encephalitogenic T cells derived from wild-type mice with active EAE. Results shown are the daily mean clinical score from two separate experiments.