. 2007 Jul-Aug;1(4 Suppl 2):S7–S12.

Table 2.

Data from five studies that have identified chromosomal gains and losses using comparative genomic hybridization techniques.^*

Recurrent chromosomal losses and deletions
	Aguirre et al⁵²	Bashyam et al⁵³	Gysin et al⁵¹	Heidenblad et al⁵⁴	Nowak et al⁵⁵
9p	X	X	X	X	X	CDKN2A, TUSC3
18q	X	X	X	X	X	Smad4
3p	X	X	X		X	FHIT, FOXP1
4q	X	X	X		X	FBXW7
6q	X	X	X		X	SEC63, SASH1, LPA
8p	X	X	X		X	DEFB103, DEFB105
17p	X	X	X		X	TP53, MKK4
6p		X	X		X
13q	X		X		X	GPC5
21q	X		X		X	PARD6G
9q				X	X	TNFSF15
10q	X			X		CDH23
12q	X			X		DUSP6
15q			X		X
21p			X		X
22q	X				X
Xp		X	X			FMR2, ARSC1
Recurrent chromosomal gains and amplifications
12p	X	X	X	X		KRAS2
20q	X		X		X	CTSZ, NCOA3
8q	X		X	X		MYC
11q	X	X	X			Cyclin D
17q	X	X	X			ERBB2
7q		X		X	X	SMURF1
5p	X		X			BASP1
7p	X		X			EGFR
14q		X			X	TGFB3
6p		X		X		NOTCH4
19q		X		X		AKT2, eIF3k
3q	X		X

Genes highlighted in boldface are known to be associated with pancreatic carcinoma.