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. 2009 Jan 26;587(Pt 6):1319–1329. doi: 10.1113/jphysiol.2008.168385

Figure 1. KCND3 5′-flanking nucleotide sequence with TRE half-site consensus and half-site consensus sequence for retinoid acid receptor (RAR) or vitamin D3 receptor (VDR) binding.

Figure 1

The point mutations G-1651T, G-707T and G-73T were introduced by site-directed mutagenesis to disrupt putative TREs.