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. 2009 Mar 6;191(9):2993–3002. doi: 10.1128/JB.01156-08

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Copyright © 2009, American Society for Microbiology

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FIG. 4. — AlgZ and AlgR control HCN production. HCN production for nonmucoid (A) and mucoid (B) algZ and algR mutants was measured as for Fig. 3. (A) PAO1, PSL317 (ΔalgR), PAZ (ΔalgZ), PAO6886 (algZ::Tc), PAO6886 harboring pUCP18Z (algZ::Tc + algZ), and PSL317Z (ΔalgR algZ::Tc). (B) PAO568, PAR568 (ΔalgR mucA2), PAZ568 (ΔalgZ mucA2), PAO6884 (algZ::Tc mucA2), PAO6884 harboring plasmid pUCP18Z (algZ::Tc mucA2 + algZ), and PAO6885 (algR::Gm algZ::Tc mucA2). Statistical significance was determined as for Fig. 3.