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. 2009 Mar 6;191(9):2993–3002. doi: 10.1128/JB.01156-08

FIG. 5.

FIG. 5.

AlgR D54 is critical for HCN. (A) HCN production was measured from WFPAO1, WFPAO1 with chromosomal algR mutation D54N (AlgR D54N), WFPAO1 with chromosomal algR mutation D85N (AlgR D85N), WFPAO1 with double chromosomal algR mutations D54N and D85N (AlgR D54N D85N), WFPAO1 containing chromosomal algR mutation D54N and an algZ deletion (D54N ΔalgZ), and WFPAO1 containing chromosomal mutations in algR D54N and D85N and an algZ deletion (D54N D85N ΔalgZ). (B) HCN production from mucoid derivatives (mucA::Tc) of the same site-directed algR and algZ mutants as those in panel A. Statistical significance was determined as described for Fig. 3.