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. 2009 Mar 9;29(10):2532–2545. doi: 10.1128/MCB.01682-08

FIG. 5.

FIG. 5.

(A) Suppression of the silencing defect of the Sir3 BAH domain mutants is due to the H3 D77N and H4 H75Y mutations. Results for six representative Sir3 mutants are depicted. Silencing was measured at HML::URA3 by growth on 5-FOA and at HMR by mating in the wild-type H3/H4 (strain VSY29), the H3 D77N mutant (VSY30), and the H4 H75Y mutant (VSY31). (B) Suppression of the silencing defect at HMR of some of the Sir3 BAH domain mutants by a histone H4 K16R mutant (strain PYY12). WT, wild type.