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. 2009 Apr 7;42(2):55–64. doi: 10.1267/ahc.08012

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© 2009 The Japan Society of Histochemistry and Cytochemistry

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 2 — Cellular localization of AK4 in the brain. (A) Immunohistochemical analyses were performed using anti-AK4. a, b; Cerebral cortex, c; Choroid plexus, d, e, f; Cerebellum. Bar=200 µm (a, d), 100 µm (b, e), and 20 µm (c, f). Py, pyramidal cell; Nc, neuroglial cell; GL, granular layer; ML, molecular layer; PL, Purkinje layer; Pu, Purkinje cell. Open square indicates the area of higher magnification. (B) Double immunodetection with anti-AK4 and neural or glial marker antibodies. Bar=100 µm.

Fig. 2 — Cellular localization of AK4 in the brain. (A) Immunohistochemical analyses were performed using anti-AK4. a, b; Cerebral cortex, c; Choroid plexus, d, e, f; Cerebellum. Bar=200 µm (a, d), 100 µm (b, e), and 20 µm (c, f). Py, pyramidal cell; Nc, neuroglial cell; GL, granular layer; ML, molecular layer; PL, Purkinje layer; Pu, Purkinje cell. Open square indicates the area of higher magnification. (B) Double immunodetection with anti-AK4 and neural or glial marker antibodies. Bar=100 µm.