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. Author manuscript; available in PMC: 2009 Dec 1.
Published in final edited form as: Neuropharmacology. 2008 Jul 23;55(7):1131–1139. doi: 10.1016/j.neuropharm.2008.07.021

Fig. 5.

Fig. 5

PICK1 and KRIP6 compete for functional regulation of recombinant GluR6. (A) Families of whole-cell currents evoked by 300 µM kainate in COS-7 cells transfected with CFP (47 cells), mRFP-PICK1 (26 cells), mRFP-PICK1 + CFP-KRIP6 (15 cells) or CFPKRIP6 (34). Curves superimposed in color show the average of currents evoked in all cells tested. (B) Peak current, steady-state/peak current, and decay time constant (Tau) are plotted as a percent of the mean values obtained with CFP alone. *Different from CFP alone (p < 0.05, t-test); #different from mRFP-PICK1 alone and from CFP-KRIP6 alone (p < 0.05, t-test), but not different from CFP alone (p > 0.05, t-test).