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. Author manuscript; available in PMC: 2009 Jun 4.
Published in final edited form as: Clin Cancer Res. 2008 Mar 1;14(5):1386–1392. doi: 10.1158/1078-0432.CCR-07-4077

Table 3.

Logistic regression models to predict NAF secretor status (N=238)

Models2
Variables1 Model 1 Model 2 Model 3A Model 3B
Lactose intake 2.3 (1.4, 3.9) 2.6 (1.4, 4.7) 2.7 (1.5, 4.8) 2.6 (1.4, 4.9)
Total caloric intake NE3 1.2 (0.6, 2.2) NE NE
Protein intake NE 0.9 (0.6, 1.2) NE NE
Fat intake NE 0.9 (0.5, 1.6) NE NE
Carbohydrate intake NE 0.9 (0.7, 1.2) NE NE
Age NE NE 1.1 (1.0, 1.2) 1.1 (1.0, 1.2)
Ethnicity NE NE 0.6 (0.3, 1.3) 0.9 (0.4, 1.8)
Menarche NE NE 0.8 (0.7, 1.0) 0.8 (0.6, 1.0)
Parity NE NE 2.3 (1.0, 5.6) NE
Age at first childbirth NE NE NE 1.5 (1.0, 2.1)
1

Lactose, increment of 10 g; total caloric intake, increment of 100 kcal; protein intake, increment of 10 g; fat intake, increment of 10 g; carbohydrate intake, increment of 10 g; age at first childbirth, increment of 5 years; parity, yes vs. no.

2

Model 1: the effect of lactose was estimated. Model 2: the effect of lactose was adjusted by other nutrients including intake of total calories, proteins, fats and carbohydrates. Model 3A: the effect of lactose was adjusted for demographic and reproductive variables, including age, ethnicity, age of menarche, and parity.

Model 3B: for parous women only (N=211), the effect of lactose was adjusted by demographical and reproductive variables, including age, ethnicity, age of menarche, and age at first childbirth.

3

NE, variable not allowed to enter the model