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. 1999 May 25;96(11):6506–6510. doi: 10.1073/pnas.96.11.6506

Figure 1.

Figure 1

SBP (A), 24-h urine sodium excretion (UNaV) (B), and 24-h urine volume (V) (C) of mice (n = 10) lacking the subtype-2 (AT2) angiotensin receptor (open bars) and WT mice (n = 10; closed bars) on normal or low sodium intake. ∗, P < 0.01 from WT mice; +, P < 0.0001 from normal sodium intake.