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. 2009 Jan;156(1):62–83. doi: 10.1111/j.1476-5381.2008.00015.x

Figure 7.

Figure 7

Influence of PDE inhibitors on the shortening of relaxation time (t1/2) by (−)-noradrenaline (100 nmol·L−1, NA) through β1-adrenoceptors (left hand panels) and (−)-adrenaline (1 µmol·L−1, Adr) through β2-adrenoceptors (right hand panels). Comparison with the responses to (−)-isoprenaline (ISO). C, control; TMC, time-matched control; Cil, cilostamide (300 nmol·L−1); Rol, rolipram (1 µmol·L−1) and IBMX (10 µmol·L−1). Data from the experiments on left ventricular papillary muscles (see Fig. 4). *P < 0.05, **P < 0.01, ***P < 0.001 vs. control; #P < 0.05 vs. catecholamine + PDE inhibitor; §P < 0.05 vs. PDE inhibitor alone.