Skip to main content
. Author manuscript; available in PMC: 2010 Jul 7.
Published in final edited form as: Neuroscience. 2009 Mar 29;161(3):718–733. doi: 10.1016/j.neuroscience.2009.03.057

Figure 4.

Figure 4

VP muscimol disrupts lick microstructure. (A) Mean (±SEM) interlick intervals occurring during consummatory bursts are shown. Relative to the control treatment (saline in both NAcc and VP), the mean interlick interval was elevated for NAcc DAMGO + VP muscimol treatment and NAcc saline + VP low muscimol treatment (asterisks, p<0.05). Overall, there was a significant elevation in ILIs comparing muscimol treatments to control saline (collapsing across DAMGO treatments, which had no effect). (B) Binned interlick interval times. Muscimol treatment shifted ILIs toward higher values. The percentage of ILIs occurring in the 0–250 ms bin was significantly decreased for muscimol treatments relative to saline control, and significantly elevated in the 251–500 ms bin (asterisks, p<0.05).