Table 7.
Study | No. of study subjects | COPD patients characteristics | BAEP parameters studied | Patients with BAEP abnormalities | BAEP parameters affected | Correlations |
---|---|---|---|---|---|---|
Kayacan et al.[4] | 32 COPD subjects [male=30]; | 19/32 had PaO2<55mmHg | Latencies I, II, III, IV, V | Individual patients data not mentioned | Latency III | III ** PEFR, FEF25, |
no controls | Age=61±8.8 years; | IPL I-III, III-V, I-V | IPL I-III, III-V | FEF25-75 | ||
smoking pack | IV** FVC, PEFR | |||||
years=37.4±28.5 | IPL I-III** | |||||
FEF25-75, FEV1/FVC, | ||||||
FEF25-75 | ||||||
IPL III-V** PaCO2, | ||||||
HCO3, pH | ||||||
Atis et al.[5] | 21 COPD patients [male=16] | Severe airflow obstruction; | Latencies I, III, V | 16/21 [76.1%] | Latency I, V | No significant |
11 had clinical neuropathy | Age=64±6.5 years; | IPL I-III, III-V, I-V | IPL III-V, I-V | Correlation with pH, PaO2, | ||
controls=21 | Only 15/21 smokers, | Amplitude wave I, III, V | PaCO2, FEV1% | |||
pack years = 24.59±21.21; | FEV1/FVC, | |||||
FEV1=0.96+0.32 | duration of disease or cigarette consumption | |||||
Our study | COPD patients =40, all male all male | Stable COPD patients, | Latencies I, II, III, IV, V | 26/40 [65%] | Latency I, III, IV, V | Left side: |
[None had clinical neurological deficiency] | Age = 57.25±9.07 | IPL I-III, III-V, I-V | IPL I-III, III-V, I-V | I, III** FEV1 | ||
Healthy volunteers=40, | Smoking pack | Amplitude I-Ia, V-Va | Amplitude I-Ia, V-Va | Amplitude I-Ia** smoking | ||
all male | years= 39.95±20.94 | Right side: | ||||
FEV1= 1.48±0.50 | Amplitude I-Ia** | |||||
Duration of illness |
Significant correlations between variables are shown by [**].