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. Author manuscript; available in PMC: 2010 May 1.
Published in final edited form as: Cell. 2009 May 1;137(3):498–508. doi: 10.1016/j.cell.2009.04.028

Figure 1. Transcriptional gene silencing of RD29A-LUC is suppressed by the rdm3-1 and rdm3-2 mutations.

Figure 1

(A) Effect of rdm3-1 and rdm3-2 on luminescence and kanamycin-resistance phenotypes in the ros1 background. Plants were grown on MS plates and subjected to luminescence imaging after cold treatment (4°C, 24 h). The plants were also grown on MS plates with kanamycin (50 μg/ml) and photographed after 10 days. (B) Northern blot analysis of RNA levels of endogenous RD29A, RD29A-LUC, and 35S-NPTII in wild type, ros1, and ros1rdm3-1. The constitutively expressed 18S rRNA was used as an RNA loading control while COR15A was used as a cold treatment control.