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. Author manuscript; available in PMC: 2010 Jun 1.
Published in final edited form as: Alcohol. 2009 Jun;43(4):277–284. doi: 10.1016/j.alcohol.2009.04.005

Fig. 3.

Fig. 3

The effect of ethanol on the τ-value of desensitization. Panel (A) depicts how ethanol (EtOH) speeded up the rate of desensitization in recording traces for all GluR-Di receptors. The peak amplitudes of the traces have been scaled to the level of 10 mM glutamate control current. (B) TARP co-expression increased EtOH’s effect on desensitization (γ2: F1,10 = 12.3, p < 0.01; γ4: F1,10 = 38.2, p < 0.001; Bonferroni test: # p < 0.05; ## p < 0.01; ### p < 0.001 for the significance of difference from GluR-Di+TARP). The effect of EtOH on desensitization was dose dependent (γ2: F4,40 = 36.6, p < 0.001; γ4: F3,30 = 34.3, p < 0.001; Dunnett’s test: * p < 0.05, *** p < 0.001). (n = 5–7)