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. Author manuscript; available in PMC: 2009 Aug 1.

Published in final edited form as: Mol Cell Biochem. 2008 Nov 1;322(1-2):53–62. doi: 10.1007/s11010-008-9939-6

A. MMP-2 protein expression in the left ventricles 3 days post-MI. Total LV lysates were analyzed by Western blot using anti-MMP-2 antibodies. Equal loading of proteins in each lane is indicated by GAPDH immunostaining. The lower panel exhibits the mean data normalized to GAPDH. MMP-2 was higher in non-infarct area, *P<0.05 vs sham; ^†P<0.05 vs WT-NI; n=3. B. MMP-9 protein expression in the left ventricle 3 days post-MI. Total LV lysates were analyzed by Western blot using anti-MMP-9 antibodies. The lower panel exhibits the mean data normalized to GAPDH. MMP-9 was higher in non-infarct area, *P<0.001 KO-INF vs sham; ^†P<0.05 vs WT-INF; n=3. NI, non-infarct area; INF, infarct area.

A. MMP-2 protein expression in the left ventricles 3 days post-MI. Total LV lysates were analyzed by Western blot using anti-MMP-2 antibodies. Equal loading of proteins in each lane is indicated by GAPDH immunostaining. The lower panel exhibits the mean data normalized to GAPDH. MMP-2 was higher in non-infarct area, *P<0.05 vs sham; ^†P<0.05 vs WT-NI; n=3. B. MMP-9 protein expression in the left ventricle 3 days post-MI. Total LV lysates were analyzed by Western blot using anti-MMP-9 antibodies. The lower panel exhibits the mean data normalized to GAPDH. MMP-9 was higher in non-infarct area, *P<0.001 KO-INF vs sham; ^†P<0.05 vs WT-INF; n=3. NI, non-infarct area; INF, infarct area.