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. 2009 Jun 1;185(5):859–874. doi: 10.1083/jcb.200812167

Figure 4.

Figure 4.

hSpindly targets both dynein and dynactin to KTs. (A) HeLa S3 cells were treated with GL2 or hSpindly siRNAs for 48 h. Nocodazole was added to the cells 2 h before they were stained with CREST serum (green), anti-p150Glued antibody (red), and DNA (blue). (B) Cells treated as in A were stained with CREST serum (green) and anti-dynein intermediate chain (DIC) antibody (red) and DNA (blue). (C) Cells were treated with GL2, hSpindly-1, or hSpindly-2 siRNAs for 48 h. Lysates from mitotic cells (nocodazole shake-off) were prepared and equal amounts of cell extracts were separated by SDS-PAGE and probed by Western blotting with the indicated antibodies. (D) Cells were transfected with hSpindly-2 siRNA together with either a myc-vector construct (V) or a myc-tagged hSpindly (siRNA-resistant) construct (WT) for 48 h. Cells were stained with anti-myc, anti-p150Glued antibodies (red), CREST serum (green), and DAPI (blue). (E) Cells were treated as in D. Lysates were prepared and equal amounts of cell extracts were separated by SDS-PAGE and probed by Western blotting with the indicated antibodies. (F) Table showing positive/negative localization of the indicated KT/centromere proteins after hSpindly depletion for 48 h (−, lost from KT; +, indistinguishable from control). For DIC and p150, experiments were repeated with treatment of nocodazole (2 h) to confirm their dependency on hSpindly. Bars, 10 µm.