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. 2009 May 15;297(1):H21–H28. doi: 10.1152/ajpheart.00292.2009

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Fig. 2. — Myocardial TLR4 mediates chemokine response to global I/R. Hearts isolated from C3H/HeN (TLR4 competent) and C3H/HeJ (TLR4 defective) mice were subjected to global I/R (20 min ischemia/60 min reperfusion). Control hearts (Perf) were perfused without subjecting to I/R. mRNA (A) and peptide (B) levels of keratinocyte-derived chemokine (KC) and monocyte chemoattractant protein-1 (MCP-1) after I/R are significantly lower in TLR4-defective hearts than in TLR4-competent hearts. Data are expressed as means ± SE; n = 5 in each group. ^aP < 0.05 vs. phenotype perfusion control; ^bP < 0.05 vs. HeN I/R.