Skip to main content
. Author manuscript; available in PMC: 2009 Oct 1.
Published in final edited form as: Nat Neurosci. 2009 Mar 22;12(4):454–462. doi: 10.1038/nn.2289

Figure 4. Pde4a−/− OSNs display no aberrant EOG response properties.

Figure 4

Data for wildtype mice is re-plotted from Figure 2 for reference. (a) Dose-response relationship of EOG responses to amyl acetate from Pde4a−/− mice (n = 5). Error bars are SD. (b) Normalized and averaged EOG responses from Pde4a−/− mice (n = 14) to a single 100 ms pulse of 10−3 M amyl acetate. Inset shows the traces plotted on an expanded time axis. (c) Termination τ for responses to three amyl acetate concentrations. For 10−4 M, P = 0.70; 10−3 M, P = 0.82; 10−2 M, P = 0.57. (d) Response latencies. For 10−4 M, P = 0.47; 10−3 M, P = 0.61; 10−2 M, P = 0.87. (e) Response rise times. For 10−4 M, P = 0.39; 10−3 M, P = 0.81; 10−2 M, P = 0.97. In (c–e), error bars are 95% CI. All P values are from unpaired t-tests, n = 14 for Pde4a−/−. No significant differences (P < 0.05) are found in any parameters.