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. 2009 May 12;100(12):1926–1936. doi: 10.1038/sj.bjc.6605072

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Copyright © 2009 Cancer Research UK

This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material.If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit https://creativecommons.org/licenses/by/4.0/.

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Prognostic significance of leading edge gene sets associated with MPRED resistance in T-ALL cell lines. Graphs show relapse-free survival in primary ALL patient cohorts (T lineage and B lineage as indicated) predicted based on expression of genes associated with MPRED resistance in T-ALL cell lines. The gene profiles represent the leading edges of the following GSEA gene sets: (A) CHOLESTEROL BIOSYNTHESIS; (B) STRING GLUL TOP100 ASSOCIATIONS (genes associated with glutamine synthetase); (C and D) V$MYC_Q2 (genes regulated by MYC); (E and F) WEI_DOWN_IN_DEX_RESISTANCE (genes associated with DEX resistance); (G) VOXPHOS (oxidative phosphorylation); (H) PENG_RAPAMYCIN_DOWN (genes downregulated in response to rapamycin). Further details of these gene sets can be found in Supplementary Table S4 and Supplementary Data File 4.