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. 2009 May 11;206(5):1149–1166. doi: 10.1084/jem.20081271

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© 2009 Lee et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.jem.org/misc/terms.shtml). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 10. — Comparisons of BRP-39 and AMCase. (A) The levels of mRNA encoding BRP-39 in mice that received antigen-specific in vitro–polarized Th1 or Th2 cells and were challenged with antigen (c, control). (B) BRP-39 expression was evaluated by RT-PCR in IFN-γ Tg mice and controls (−). (C) AMCase and BRP-39 expression were localized in the lungs of 2-mo-old IL-13 Tg mice by double-label IHC (white arrows, colocalized; yellow arrows, AMCase; red arrows, BRP-39). (D and E) The levels of BAL AMCase were assessed in OVA-plus-alum–sensitized and OVA-challenged mice (D) and IL-13 Tg mice with WT and null BRP-39 loci (E). The values in D and E are the mean ± SEM of evaluations in a minimum of five animals and are representative of two separate evaluations. *, P < 0.05. Bars, 25 µm.