Table 2.
T-DNA insertions and de-etiolation phenotype in different lines of SALK_024018
| oep16.1–1 | arogp1–1 | cm1–1 | det-p, % | n | |
|---|---|---|---|---|---|
| Col-0 | wt | wt | wt | 14.7 | 265 |
| flu | wt | wt | wt | 31.6* | 196 |
| F6–4a | ho | wt | wt | 70.7* | 215 |
| 5.2 | ho | ho | ho | 85.8* | 190 |
| 5.10 | ho | ho | ho | 49.2* | 130 |
| 4.1 | ho | ho | wt | 15.0 | 233 |
| 4.2 | wt | ho | wt | 46.9* | 177 |
| 19.3 | wt | wt | wt | 48.4* | 182 |
| 2.2 | wt | wt | ho | 5.1 | 217 |
Different lines segregating from SALK_024018, which displayed variable quantities of a de-etiolation phenotype (det-p) as described in Table 1, were PCR genotyped for the T-DNA insertions in OEP16.1, AroGP1, and CM1. wt, wild type; ho, homozygous for the respective mutant allele.
*, Lines with >30% dead seedlings (compare heterozygous flu control) were considered to show a de-etiolation phenotype.