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. Author manuscript; available in PMC: 2009 Jul 25.
Published in final edited form as: Mol Cell Biochem. 2007 Dec 13;311(1-2):1–7. doi: 10.1007/s11010-007-9686-0

Figure 1.

Figure 1

Left panel: Dose-response to Ang-I (10−10 to 10−7 mol/L) and diameter decrease relationship in normotensive (NT) and hypertensive (HT) mouse mesenteric resistance artery held at 50 mmHg without and with ACE inhibitor (captopril, 10 μM), n=6 per group. *P<0.05, statistically significant, NT vs. NT+captopril; HTN vs. HTN+captopril; Right panel: Dose-response to Ang-I (10−10 to 10−7 mol/L) and diameter decrease relationship in normotensive (NT) and hypertensive (HT) mouse mesenteric resistance artery held at 50 mmHg without and with intraluminal specific ACE antibody (50 ng/ml), n=6 per group. *P<0.05, statistically significant, NT vs. NT+ACE antibody at 10−7 of Ang-I