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. Author manuscript; available in PMC: 2010 Jan 1.
Published in final edited form as: J Immunol. 2009 Jan 1;182(1):623–635. doi: 10.4049/jimmunol.182.1.623

Figure 3. Intratracheal transfer of Th2 cells does not restore eosinophil trafficking into the airways in STAT6-/- mice.

Figure 3

A) Representative flow cytometry of BAL cells stained for CD4 and KJ following intratracheal transfer of OVA-specific in vitro polarized Th2 lymphocytes and 3 OVA challenges (n = 6 mice per group, from 2 experiments). B) Number of CD4+ and KJ+ OVA-specific Th2 cells in the BAL of wild-type and STAT6-/- mice following intratracheal transfer of OVA-specific in vitro polarized Th2 lymphocytes and 3 OVA challenges (n = 6 mice per group, from 2 experiments). C) Number of eosinophils in the BAL of wild-type and STAT6-/-mice following intratracheal transfer of OVA-specific in vitro polarized Th2 lymphocytes and 3 OVA challenges (n = 6 mice per group, from 2 experiments). D) Percentage of CCR3+ cells in the granulocyte gate of cells isolated from blood of wild-type and STAT6-/- mice following intratracheal transfer of OVA-specific in vitro polarized Th2 lymphocytes and 3 OVA or PBS challenges (n = 3 mice per group, from 1 experiment). E) Lung chemokine RNA copies normalized to copies of GAPDH RNA in wild-type and STAT6-/- mice following intratracheal transfer of OVA-specific in vitro polarized Th2 lymphocytes and 3 OVA challenges (n = 6 mice per group, from 2 experiments).