. Author manuscript; available in PMC: 2009 Aug 10.

Published in final edited form as: Oncogene. 2008 Jul 28;27(51):6561–6570. doi: 10.1038/onc.2008.253

Table 3.

E2f1;E2f3b double mutant animals arise at the expected frequency

Cross conducted	E2f1^-/-;E2f3b^-/- observed	Expected
E2f1^+/-;E2f3b^-/-x E2f1^-/-;E2f3b^+/-	25% (6/25)	25%
E2f1^-/-;E2f3b^+/-x E2f1^+/-;E2f3b^+/-	12.5% (2/16)	12.5%
E2f1^+/-;E2f3b^-/-x E2f1^+/-;E2f3b^+/-	16.7% (3/18)	12.5%
E2f1^-/-;E2f3b^-/-x E2f1^+/-;E2f3b^+/-	23% (3/13)	25%
E2f1^-/-;E2f3b^-/-x E2f1^+/-;E2f3b^-/-	40% (2/5)	50%
χ²test p value (sum)		0.98

The indicated crosses were performed using mice on a C57BL/6 × 129Sv background and the pups genotyped at three weeks of age. The frequency of double mutant animals is shown and was determined not to be statistically significantly different from the expected frequency using a χ² test.