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. Author manuscript; available in PMC: 2009 Aug 10.
Published in final edited form as: J Comp Neurol. 2008 Apr 10;507(5):1780–1794. doi: 10.1002/cne.21650

Fig. 3.

Fig. 3

Olfactory bulb tyrosine hydroxylase (TH) expression is absent in Sall3-deficient animals. Reelin (A,B) and TH (C–F) staining (black) in coronal sections of control (A,C,E) and Sall3 mutant (B,D,F) animals at P0.5 (A–D) and E16.5 (E,F). Sections were counterstained with hematoxylin and eosin. No alterations in reelin expression in the MCL were observed in Sall3-deficient animals (B). TH expression in the GL was absent in Sall3 mutant animals at P0.5 (D) and E16.5 (F). GCL, granule cell layer; MCL, mitral cell layer; GL, glomerular layer; ON, olfactory nerve. Scale bar = 100 µm.