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. 2009 Jun 2;8(8):3899–3911. doi: 10.1021/pr900185s

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Copyright © 2009 American Chemical Society

This is an open-access article distributed under the ACS AuthorChoice Terms & Conditions. Any use of this article, must conform to the terms of that license which are available at http://pubs.acs.org.

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Cluster analysis based on E. caproni infection-induced metabolic interactions between 8 assessed biological matrices. (A) Correlation structure of control animals demonstrating natural variation due to genetic and experimental individual background; (B) correlation structure composed of inherent individual variation and the effects of an infection; (C) differential cluster matrix showing only the remaining effects of the E. caproni infection. Key: co, colon; fe, fecal water; il, ileum; je, jejunum; ki, kidney; li, liver; pl, plasma; ur, urine. Statistically significant pairwise correlations between metabolite levels with p < 0.05 were explored in the analysis.

Cluster analysis based on E. caproni infection-induced metabolic interactions between 8 assessed biological matrices. (A) Correlation structure of control animals demonstrating natural variation due to genetic and experimental individual background; (B) correlation structure composed of inherent individual variation and the effects of an infection; (C) differential cluster matrix showing only the remaining effects of the E. caproni infection. Key: co, colon; fe, fecal water; il, ileum; je, jejunum; ki, kidney; li, liver; pl, plasma; ur, urine. Statistically significant pairwise correlations between metabolite levels with p < 0.05 were explored in the analysis.