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. 2009 Jan 19;132(3):801–809. doi: 10.1093/brain/awn355

Table 2.

Main features of CAFSA compared with known free sialic acid disorders

CAFSA Salla disease (Aula and Gahl, 2001) ISSD (Lemyre et al., 1999) Sialuria (Aula and Gahl, 2001)
Age at onset 10–24 years Infancy to Childhood 1st year of life Infancy
Horizontal nystagmus No Common Yes No
Cerebellar ataxia Yes Yes Yes No
Pyramidal syndrome No Yes ND No
Cognitive abnormalities Cognitive or behavioural decline Psychomotor retardation Psychomotor retardation (severe) Psychomotor retardation (mild)
Peripheral neuropathy Axonal > demyelinating 50% (demyelinating) ND ND
Dysmorphism No Mild Yes Yes
Growth retardation Possible Yes Yes No
Signs of organ storage No No Yes Yes
Cerebral MRI
    Cerebellar atrophy Mild to moderate Moderate to severe Severe No
    Thin corpus callosum No Yes Yes No
    White matter abnormality Hilus of dentate nucleus, peridentate white matter and periventricular Diffuse hypomyelination Diffuse hypomyelination No
Free sialic acid elevation
    Urine No Common*(+) Yes (++) Yes (+++)
    CSF Yes Yes ND ND
    Fibroblasts No Yes (lysosomal) Yes (lysosomal) Yes (cytoplasmic)

*Salla patients have been reported without sialuria (Mochel et al., Ann Neurol, in press). ISSD = Infantile free sialic storage disease; ND = not determined; + = mild; ++ = moderate; +++ = massive.