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. 2009 May 5;587(Pt 13):3271–3285. doi: 10.1113/jphysiol.2009.169771

Figure 2. Systemic (in vivo) EDD to ACh (n= 6 per group) (A) and EID to sodium nitroprusside (SNP) (B), carotid artery EDD to ACh in the absence or presence of the NOS inhibitor, l-NAME (n= 6–10 per group) (C), and carotid artery EID to SNP in young (Y) and old (O) cage control (CC) and voluntary wheel running mice (VR) (D).

Figure 2

Values are means ±s.e.m.*P < 0.05 vs. YCC.