Table 2.

Quantification of process defects in mig-32 mutants

Genotype	Cell process	Animals with a defect (%)	n
Pnlp-1gfp	HSN axon	0	50
mig-32(n4275), Pnlp-1gfp	HSN axon	56	49
mig-32(tm1684), Pnlp-1gfp	HSN axon	46	46
mig-32(tm1807), Pnlp-1gfp	HSN axon	56	88
Psra-6gfp	PVQ axons	8	50
mig-32; Psra-6gfp	PVQ axons	26	87
Punc-47gfp	VD commissures	4	25
mig-32; Punc-47gfp	VD commissures	63	100

The percentage of animals with a defect in specific neuronal processes is shown. For the HSN axons, animals were scored as defective if the axon failed to reach the head; in general, axons that reached the head followed a normal path from the HSN ventrally into the ventral nerve cord then turned anterior to the head. HSN neurons that failed to migrate to the midbody often had more severe defects in axon pathfinding, with axons that tracked posterior rather than anterior. For the PVQ axons, defective axons included those that crossed the midline inappropriately, as compared with wild-type animals. For the VD commissures, defective commissures included those that tracked on the wrong side of the body wall. mig-32 mutants had 0-5 commissures on the wrong side, with posterior VD neurons being more likely to have defective commissures