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Enhanced activity within the ventral hippocampus results in an increased activation of the nucleus accumbens (NAc). The subsequent increase in NAc output inhibits the ventral pallidum (VP) resulting in the disinhibition of VTA dopamine neurons. This enhanced mesolimbic dopamine neuron population activity is suggested to underlie the augmented response to amphetamine in MAM-treated rats and psychosis in human schizophrenia patients.
