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. Author manuscript; available in PMC: 2010 Sep 1.
Published in final edited form as: J Neurochem. 2009 Jun 22;110(5):1409–1421. doi: 10.1111/j.1471-4159.2009.06232.x

Figure 7. The induction of splenic leukocyte apoptosis after T3 SCI is attenuated using antagonists of GRs and β2ARs.

Figure 7

(A–C) Spleen weight (A), total numbers of splenocytes and circulating white blood cell counts (B&C, respectively) are reduced after T3 SCI (3 days post injury). These SCI-specific effects were partially reversed by co-administration of butoxamine (Butox; a selective β2AR antagonist) and RU486 (a GR antagonist). n=5/group; ***p<0.001, **p<0.01, *p<0.05 as indicated in A&B or vs. laminectomy control in C; One-way ANOVA with Tukey’s Post-test.