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. 2009 Jul 16;28(17):2636–2649. doi: 10.1038/emboj.2009.204

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Copyright © 2009, European Molecular Biology Organization

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Contribution of essential and non-essential L-AChR subunits to cholinergic agonist sensitivity in nra-2(ok1731) mutants. (A, B) Paralysis assays (n=2–7; 30 animals each) in response to levamisole (A) and nicotine (B) of mutants in nra-2(ok1731), lev-8(x15), acr-8(ok1240) and unc-38(x20), and in double-mutant combinations as indicated. (C) Normalized mean peak values of ACh-, levamisole- and nicotine-induced muscle PSCs in wild-type animals, nra-2(ok1731) mutants, and mutants of the non-essential L-AChR α-subunits lev-8(x15) and acr-8(ok1240) as well as respective double mutants. Displayed are means±s.e.m., number of animals and significant differences to wild type (t-test; ^*P<0.05; ^**P<0.01; ^***P<0.001) are indicated.