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NFATc3 genetic ablation prevents CH-mediated increases in expression of sGC-α1 in mouse pulmonary arteries. Data are fold change in mRNA (A) and protein (B) normalized to β-actin in pulmonary arteries from NFATc3 wild-type (WT) and knockout (KO) mice exposed to N or CH for 2d or 21d. Values are means ± SE; n = 6. *P < 0.05 vs. N WT and CH 21d WT. #P < 0.05 vs. N WT.
